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Kan CBDA reglera transkriptionsaktiviteten hos pro-inflammatoriska gener?

Kan CBDA reglera transkriptionsaktiviteten hos pro-inflammatoriska gener? CBDA kan reglera transkriptionsaktiviteten hos pro-inflammatoriska gener genom att modulera transkriptionsfaktorer som NF-KB eller AP-1. Dessa transkriptionsfaktorer kontrollerar uttrycket av gener involverade i inflammation, och deras överaktivering bidrar till kroniska inflammatoriska sjukdomar. Genom att hämma aktiveringen av dessa faktorer kan CBDA minska uttrycket av pro-inflammatoriska gener och dämpa inflammation. Detta tyder på att CBDA kan vara ett användbart terapeutiskt medel för inflammatoriska tillstånd. Det behövs dock fler studier för att bekräfta CBDA:s effekter på pro-inflammatorisk gentranskription.

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