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Hur skiljer sig CBG:s interaktion med CB1- och CB2-receptorer från THC och CBD?

Hur skiljer sig CBG:s interaktion med CB1- och CB2-receptorer från THC och CBD? CBG interagerar med CB1- och CB2-receptorer annorlunda än THC och CBD. CBG har låg affinitet för CB1-receptorer, vilket betyder att det inte producerar de psykoaktiva effekter som är förknippade med THC. Istället fungerar den som en partiell agonist vid både CB1- och CB2-receptorer, och modulerar endocannabinoidaktivitet utan överstimulering. Till skillnad från CBD, som har en minimal interaktion med CB1-receptorer, hjälper CBG:s balanserade interaktion med både CB1- och CB2-receptorer till att reglera humör, smärta och inflammation. Detta distinkta interaktionsmönster gör CBG användbart för att behandla tillstånd som ångest, inflammation och neurodegeneration utan att orsaka den höga associerade med THC.

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