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W jaki sposób CBG oddziałuje z jonotropowymi receptorami glutaminianu w neuroprotekcji?

CBG oddziałuje z jonotropowymi receptorami glutaminianu, szczególnie z receptorami NMDA, w celu zmniejszenia ekscytotoksyczności – procesu, w którym nadmiar glutaminianu prowadzi do uszkodzenia neuronów. Hamuje nadmierną aktywację tych receptorów, zapobiegając nadmiernemu napływowi wapnia do neuronów. Działanie to chroni przed uszkodzeniem i śmiercią neuronów, co ma kluczowe znaczenie w chorobach neurodegeneracyjnych, takich jak choroby Alzheimera i Parkinsona. Dzięki tej modulacji CBG pomaga utrzymać zdrowie i funkcjonowanie neuronów, oferując potencjalne korzyści neuroprotekcyjne.

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