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W jaki sposób CBDA reguluje wewnątrzkomórkowy handel mediatorami stanu zapalnego?

W jaki sposób CBDA reguluje wewnątrzkomórkowy handel mediatorami stanu zapalnego? CBDA może regulować wewnątrzkomórkowy handel mediatorami stanu zapalnego poprzez modulowanie komórkowych szlaków sygnałowych zaangażowanych w transport pęcherzykowy i wydzielanie cytokin. Może to obejmować wpływ na ruch cytokin, chemokin i innych mediatorów stanu zapalnego do i z przedziałów komórkowych, takich jak pęcherzyki i lizosomy. Modulując handel, CBDA może zmniejszyć uwalnianie cząsteczek prozapalnych do krążenia, kontrolując w ten sposób stan zapalny. Efekt ten może być korzystny w leczeniu chorób, w których nadmierne uwalnianie mediatorów stanu zapalnego przyczynia się do patologii. Potencjał CBDA do regulowania tego procesu może zapewnić kolejny mechanizm jego działania przeciwzapalnego.

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