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Hvordan modulerer CBN uttrykket av pro-inflammatoriske cytokiner i autoimmune sykdomsmodeller?

Hvordan modulerer CBN uttrykket av pro-inflammatoriske cytokiner i autoimmune sykdomsmodeller? CBN modulerer pro-inflammatoriske cytokiner ved å interagere med CB2-reseptorer på immunceller, og undertrykker frigjøringen av cytokiner som TNF-α og IL-6. Denne antiinflammatoriske virkningen reduserer overaktivering av immunforsvaret og vevsskader som vanligvis sees ved autoimmune sykdommer. CBN reduserer også oksidativt stress, reduserer betennelse ytterligere og fremmer vevsreparasjon. Disse egenskapene tyder på at CBN kan være effektiv i å håndtere autoimmune tilstander som revmatoid artritt eller multippel sklerose. Dens doble rolle i betennelseskontroll og immunmodulering gjør den til en lovende terapeutisk kandidat, selv om ytterligere forskning er nødvendig for å validere disse effektene hos mennesker.

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