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Hvordan påvirker CBN reguleringen av genuttrykk relatert til betennelse og oksidativt stress?

Hvordan påvirker CBN reguleringen av genuttrykk relatert til betennelse og oksidativt stress? CBN påvirker genuttrykk relatert til betennelse og oksidativt stress ved å modulere aktiviteten til transkripsjonsfaktorer og signalveier som regulerer disse prosessene. Gjennom aktivering av CB2-reseptorer reduserer CBN uttrykket av pro-inflammatoriske cytokiner og andre markører for betennelse. Det øker også uttrykket av gener som er involvert i antioksidantforsvaret, og bidrar til å beskytte cellene mot oksidativ skade. Denne doble handlingen – reduserer betennelse samtidig som den fremmer antioksidantresponser – gjør CBN til et verdifullt terapeutisk middel for tilstander assosiert med kronisk betennelse og oksidativt stress, som autoimmune sykdommer og nevrodegenerative lidelser.

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